In: Diabetologia, 2015, vol. 58, no. 8, p. 1827-1835
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In: The Journal of Clinical Endocrinology & Metabolism, 2016, vol. 101, no. 8, p. 3240-3249
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In: Diabetologia, 2015, vol. 58, no. 3, p. 456-463
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In: Diabetologia, 2015, vol. 58, no. 2, p. 304-312
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In: Human Molecular Genetics, 2017, vol. 26, no. 18, p. 3453-3465
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In: Diabetologia, 2015, vol. 58, no. 2, p. 221-232
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In: iScience, 2019, vol. 19, p. 39–53
Insulin-induced AKT activation is dependent on phosphoinositide 3-kinase and opposed by tumor suppressor phosphatase and tensin homolog (PTEN). Our previous study demonstrates that myosin 1b (MYO1B) mediates arginase-II-induced activation of mechanistic target of rapamycin complex 1 that is regulated by AKT. However, the role of MYO1B in AKT activation is unknown. Here we show that silencing...
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In: Journal of Pediatric Endocrinology and Metabolism, 2015, vol. 28, no. 5-6, p. 539-544
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In: Pflügers Archiv - European Journal of Physiology, 2009, vol. 457, no. 4, p. 931-940
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In: Diabetologia, 2008, vol. 51, no. 8, p. 1429-1439
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