In: Journal of Neurology, 2015, vol. 262, no. 4, p. 870-880
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In: EMBO molecular medicine, 2017, vol. 9, no. 8, p. 1000–1010
The neuromuscular junction has retained through evolution the capacity to regenerate after damage, but little is known on the inter‐cellular signals involved in its functional recovery from trauma, autoimmune attacks, or neurotoxins. We report here that CXCL12α, also abbreviated as stromal‐derived factor‐1 (SDF‐1), is produced specifically by perisynaptic Schwann cells following ...
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In: PLOS genetics, 2019, vol. 15, no. 4, p. e1008069
In the peripheral nervous system (PNS) myelinating Schwann cells synthesize large amounts of myelin protein zero (P0) glycoprotein, an abundant component of peripheral nerve myelin. In humans, mutations in P0 cause the demyelinating Charcot-Marie-Tooth 1B (CMT1B) neuropathy, one of the most diffused genetic disorders of the PNS. We previously showed that several mutations, such as the...
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In: Cell Reports, 2019, vol. 27, no. 11, p. 3152-3166.e7
After a peripheral nerve lesion, distal ends of injured axons disintegrate into small fragments that are subsequently cleared by Schwann cells and later by macrophages. Axonal debris clearing is an early step of the repair process that facilitates regeneration. We show here that Schwann cells promote distal cut axon disintegration for timely clearing. By combining cell-based and in vivo...
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In: Journal of Molecular Neuroscience, 2008, vol. 35, no. 1, p. 91-100
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In: Cellular and Molecular Life Sciences, 2007, vol. 64, no. 24, p. 3261-3265
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In: Pediatric Radiology, 1997, vol. 27, no. 2, p. 155-158
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In: Journal of Molecular Medicine, 2006, vol. 84, no. 7, p. 532-543
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In: Acta Neuropathologica, 2007, vol. 114, no. 2, p. 175-185
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In: Molecular Neurobiology, 2011, vol. 44, no. 3, p. 303-312
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