In: Sleep, 2016, vol. 39, no. 6, p. 1249-1252
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In: Cerebral Cortex, 2016, vol. 26, no. 8, p. 3553-3562
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In: Psychopharmacology, 2015, vol. 232, no. 16, p. 2981-2994
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In: SLEEP, 2016, vol. 40, no. 3, p. -
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Thèse de doctorat : Université de Fribourg, 2020.
Research on clinical populations with eating disturbances has shown the involvement of reward processing in the maintenance of disease- associated symptoms. Investigation on food as a reward indicates the metabolic or feeding state (i.e. fasted or fed state) as a modulator of food as a reward, but little is known about how these states influence other types of reward. This is important...
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In: Appetite, 2019, vol. 134, p. 26-33
BACKGROUND: Previous studies using neuroimaging and behavioral measures reported altered reward processing in anorexia nervosa (AN). In addition, anhedonia states are frequently reported in AN, potentially due to the physiological stress produced by the permanent starvation. We investigated the effect of fasting and satiety on mood and reaction times to monetary rewards in AN patients and healthy...
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In: European Eating Disorders Review, 2020, vol. 28, no. 1, p. 46-54
Anorexia nervosa (AN) is an eating disorder characterized by a low food intake and often exceeding exercise, leading to a particularly low body × weight proportion. Patients with AN usually report less hunger than healthy controls. Endogenous endocannabinoids (eCBs), specifically the anandamide, have been associated to hunger, as a meal initiator, but research regarding AN and eCB and...
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In: Frontiers in Psychology, 2019, p. 1-5
Impaired decision-making under conditions of uncertainty seems to contribute to theexpression and maintenance of anorexia nervosa (AN), but it is not clear whether thisimpairment is a disease state that would remit with treatment, or a persisting trait inpatients with AN. To examine this question, a longitudinal study was conducted in12 female inpatients with AN (age M = 22.2, SE = 1.36), before...
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In: Nature communications, 2017, vol. 8, p. 1917
The melastatin-like transient-receptor-potential-7 protein (TRPM7), harbouring a cation channel and a serine/threonine kinase, has been implicated in thymopoiesis and cytokine expression. Here we show, by analysing TRPM7 kinase-dead mutant (Trpm7 R/R) mice, that the enzymatic activity of the receptor is not essential for thymopoiesis, but is required for CD103 transcription and gut-homing of ...
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In: Hormone Molecular Biology and Clinical Investigation, 2016, vol. 28, no. 1, p. 39-53
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