In: Experimental Brain Research, 2015, vol. 233, no. 5, p. 1365-1375
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In: Cerebral Cortex, 2017, vol. 27, no. 6, p. 3217-3230
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In: European Archives of Psychiatry and Clinical Neuroscience, 2015, vol. 265, no. 8, p. 637-645
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In: International Journal of Neuropsychopharmacology, 2016, vol. 19, no. 11, p. -
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In: Social Cognitive and Affective Neuroscience, 2018, vol. 13, no. 10, p. 1071-1080
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In: Schizophrenia Bulletin, 2018, vol. 44, no. 1, p. 147-157
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In: Schizophrenia Bulletin, 2018, vol. 44, no. 1, p. 182-192
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In: Schizophrenia Bulletin, 2017, vol. 43, no. 3, p. 493-496
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In: Frontiers in Cellular Neuroscience, 2020, vol. 14, p. -
In neurodevelopmental disorders (NDDs) including autism spectrum disorder (ASD) and schizophrenia, impairment/malfunctioning of a subpopulation of interneurons expressing the calcium-binding protein parvalbumin (PV) –here termed Pvalb neurons– has gradually emerged as a possible cause. These neurons may represent a hub or point-of-convergence in the etiology of NDD. Increased oxidative...
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In: NeuroImage: Clinical, 2020, vol. 26, p. 102193
Introduction: Anhedonia, a core symptom of Major Depressive Disorder (MDD), manifests as a lack or loss of motivation as reflected by decreased reward responsiveness, at both behavioral and neural (i.e., striatum) levels. Exposure to stressful life events is another important risk factor for MDD. However, the mechanisms linking reward-deficit and stress to MDD remain poorly understood. Here,...
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