In: International Journal of Public Health, 2015, vol. 60, no. 7, p. 753-754
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In: International Journal of Public Health, 2015, vol. 60, no. 8, p. 999-1000
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In: Annals of Surgical Oncology, 2015, vol. 22, no. 13, p. 4198-4203
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In: Glycobiology, 2017, vol. 27, no. 6, p. 525-535
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In: International Journal of Public Health, 2015, vol. 60, no. 6, p. 631-632
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In: International Journal of Public Health, 2015, vol. 60, no. 7, p. 757-759
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In: Scientific Reports, 2020, vol. 10, no. 1, p. 8268
Sphingosine-1-phosphate is a signaling molecule involved in the control of cell migration, differentiation, survival and other physiological processes. This sphingolipid metabolite can be degraded by the action of sphingosine-1-phosphate lyase (SPL) to form hexadecenal and ethanolamine phosphate. The importance of SPL-mediated ethanolamine phosphate formation has been characterized in only...
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In: International journal of environmental research and public health, 2018, vol. 15, no. 5, p. 918
Identifying correlates of sedentary behaviour across all levels of the ecological model and understanding their interrelations is a promising method to plan effective interventions. The present study examined whether the objectively assessed and the perceived neighbourhood are associated with children’s sedentary behaviour time (SBT). A comprehensive set of factors at different levels of...
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In: Frontiers in Immunology, 2019, vol. 10, p. -
T cell activation in lymphoid tissue occurs through interactions with cognate peptide- major histocompatibility complex (pMHC)-presenting dendritic cells (DCs). Intravital imaging studies using ex vivo peptide-pulsed DCs have uncovered that cognate pMHC levels imprint a wide range of dynamic contacts between these two cell types. T cell—DC interactions vary between transient,...
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In: Cell reports, 2017, vol. 18, no. 10, p. 2427-2440
The mechanism by which inflammation influences the adaptive response to vaccines is not fully understood. Here, we examine the role of lymph node macrophages (LNMs) in the induction of the cytokine storm triggered by inactivated influenza virus vaccine. Following vaccination, LNMs undergo inflammasome-independent necrosis-like death that is reliant on MyD88 and Toll-like receptor 7 (TLR7)...
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