In: Angiogenesis, 2015, vol. 18, no. 3, p. 327-345
|
In: SLEEP, 2016, vol. 40, no. 3, p. -
|
In: European Journal of Clinical Microbiology & Infectious Diseases, 2015, vol. 34, no. 3, p. 461-465
|
In: Pathogens, 2020, vol. 9, no. 1, p. 21
Microglia are the chief immune cells of the brain and have been reported to be activated in severe malaria. Their activation may drive towards neuroinflammation in cerebral malaria. Malaria-infected red blood cell derived-extracellular vesicles (MiREVs) are produced during the blood stage of malaria infection. They mediate intercellular communication and immune regulation, among other...
|
In: Arthritis research & therapy, 2018, vol. 20, p. 114
Background: B cells exert their pathogenic action in rheumatoid arthritis (RA) locally in the synovium. This study was undertaken to elucidate the chemokines responsible for the recruitment of B cells in the inflamed synovium, taking into account that the rich chemokine milieu present in the synovial tissue can fine-tune modulate discrete chemokine receptors. Methods: Expression levels of...
|
In: Frontiers in immunology, 2018, vol. 9, p. 2118
Chemokine synergy-inducing molecules are emerging as regulating factors in cell migration. The alarmin HMGB1, in its reduced form, can complex with CXCL12 enhancing its activity on monocytes via the chemokine receptor CXCR4, while the form containing a disulfide bond, by binding to TLR2 or TLR4, initiates a cascade of events leading to production of cytokines and chemokines. So far, the...
|
In: Clinical and Translational Oncology, 2014, vol. 16, no. 1, p. 85-90
|
In: Cancer Immunology, Immunotherapy, 2014, vol. 63, no. 3, p. 247-257
|
In: Journal of The American Society for Mass Spectrometry, 2014, vol. 25, no. 6, p. 1083-1086
|
In: Graefe's Archive for Clinical and Experimental Ophthalmology, 2014, vol. 252, no. 9, p. 1477-1481
|