In: American Journal of Hypertension, 1988, vol. 1, no. 2, p. 193-199
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In: British Journal of Anaesthesia, 2004, vol. 93, no. 1, p. 53-62
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In: Nephrology Dialysis Transplantation, 2001, vol. 16, p. 19-25
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In: British Journal of Anaesthesia, 2003, vol. 91, no. 4, p. 525-531
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In: American Journal of Hypertension, 1999, vol. 12, no. S4, p. 205A-205A
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In: Hypertension, 2010, p. -
The activation of the sympathetic nervous system through the central actions of the adipokine leptin has been suggested as a major mechanism by which obesity contributes to the development of hypertension. However, direct evidence for elevated sympathetic activity in obesity has been limited to muscle. The present study examined the renal sympathetic nerve activity and cardiovascular effects of a...
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In: International Journal of Obesity, 2007, vol. 31, no. 2, p. 378–381
Mice lacking β-adrenoceptors, which mediate the thermogenic effects of norepinephrine and epinephrine, show diminished thermogenesis and high susceptibility to obesity, whereas mice lacking stearoyl-CoA desaturase 1 (SCD1), which catalyzes the synthesis of monounsaturated fatty acids, show enhanced thermogenesis and high resistance to obesity. In testing whether β-adrenergic...
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