In: Cellular and Molecular Life Sciences CMLS, 2005, vol. 62, no. 2, p. 179-187
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In: Molecular Biotechnology, 2004, vol. 28, no. 2, p. 147-166
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In: Journal of Gastrointestinal Surgery, 2005, vol. 9, no. 1, p. 69-74
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In: International Immunology, 2004, vol. 16, no. 1, p. 139-148
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In: Molecular Human Reproduction, 1999, vol. 5, no. 11, p. 1077-1082
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In: European Cytokine Network, 2007, vol. 18, no. 2, p. 39-45
To study the role of the redox state regulator glutathione (GSH) in bacterial lipopolysaccharide (LPS)-induced anorexia we measured GSH in liver, serum and brain in response to intraperitoneal (ip) lipopolysaccharide (LPS, 4μg/mouse) injection in LPS-naïve and LPS-pretreated (4 μg/mouse) mice. LPS reduced food intake in LPS-naïve mice and LPS pretreatment attenuated this effect. LPS...
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In: European Cytokine Network, 2007, vol. 18, no. 2, p. 47-55
Since the redox state regulator glutathione (GSH) influences lipopolysaccharide (LPS) anorexia, we studied the roles of tumour necrosis factor-alpha (TNFα) and nerve growth factor (NGF) in the GSH response to intraperitoneal (ip) LPS injection in mice. Basal NGF and GSH levels were up-regulated in brain and liver of TNFα-knock-out (KO) mice, and this was associated with attenuated LPS anorexia....
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