In: Forensic Science, Medicine, and Pathology, 2013, vol. 9, no. 4, p. 501-505
|
In: European Journal of Cardio-Thoracic Surgery, 2008, vol. 34, no. 3, p. 653-660
|
In: Cardiovascular Research, 2007, vol. 76, no. 3, p. 482-493
|
In: Glycobiology, 2000, vol. 10, no. 2, p. 141-148
|
In: European Heart Journal, 2005, vol. 26, no. 21, p. 2334-2343
|
In: European Journal of Cardio-Thoracic Surgery, 2011, vol. 39, no. 5, p. e139-e143
|
In: Journal of Vascular Surgery, 2009, vol. 50, no. 1, p. 161-170
Objective: Mitogen-activated protein kinases (MAPKs), including JNK, p38, and ERK1/2, noticeably influence ischemia/reperfusion injury (IRI). The complement inhibitor dextran sulfate (DXS) associates with damaged endothelium denudated of its heparan sulfate proteoglycan (HSPG) layer. Other glycosaminoglycan analogs are known to influence MAPK signaling. Hypothetically therefore, targeted...
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