In: Nutrients, 2020, vol. 12, no. 11, p. 10 p
Metabolic alkalosis may develop as a consequence of urinary chloride (and sodium) wasting, excessive loss of salt in the sweat, or intestinal chloride wasting, among other causes. There is also a likely underrecognized association between poor salt intake and the mentioned electrolyte and acid–base abnormality. In patients with excessive loss of salt in the sweat or poor salt intake, the...
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In: Pflügers Archiv : European Journal of Physiology, ///-
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In: Pediatric Nephrology, 2015, vol. 30, no. 9, p. 1389-1396
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In: Journal of Cellular and Molecular Medicine, 2019, vol. 23, no. 10, p. 6543–6553
The NPHS2 gene, encoding the slit diaphragm protein podocin, accounts for genetic and sporadic forms of nephrotic syndrome (NS). Patients with NS often present symptoms of volume retention, such as oedema formation or hypertension. The primary dysregulation in sodium handling involves an inappropriate activation of the epithelial sodium channel, ENaC. Plasma proteases in a...
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In: Biogerontology, 2005, vol. 6, no. 5, p. 325-334
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In: Virchows Archiv, 2008, vol. 452, no. 5, p. 499-505
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In: Cancer Causes & Control, 2011, vol. 22, no. 10, p. 1375-1382
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In: Urological Research, 1997, vol. 25, p. S25-S30
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In: European Radiology, 2013, vol. 23, no. 10, p. 2899-2905
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In: Diabetologia, 2010, vol. 53, no. 8, p. 1568-1575
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