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- Inglese (23)
Autore
- Loffing, Johannes (23)
- Loffing-Cueni, Dominique (9)
- Devuyst, Olivier (4)
- Kaissling, Brigitte (4)
- Gailly, Philippe (3)
- Rossier, Bernard C. (3)
- Schwaller, Beat (3)
- Ackermann, Daniel (2)
- Carrel, Monique (2)
- Cueni-Loffing, Dominique (2)
- Daidié, Dorothée (2)
- Di Chiara, Marianna (2)
- Flores, Sandra Y. (2)
- Gresko, Nikolay (2)
- Hegyi, Ivan (2)
- Hummler, Edith (2)
- Le Hir, Michel (2)
- Olinger, Eric (2)
- Sauter, Daniel (2)
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- Verrey, François (2)
- Vogt, Bruno (2)
- Wehrli, Philipp (2)
- Auberger, Ines (1)
- Aydin, Selda (1)
- Beatrice, Rohrbach (1)
- Beauwens, Renaud (1)
- Belge, Hendrica (1)
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- Bindels, René J. (1) Di più Meno
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Parola chiave
- Aldosterone (4)
- ENaC (3)
- NCC (3)
- Amiloride (2)
- DCT (2)
- Epithelial sodium channel (2)
- FSP1 (2)
- Gitelman syndrome (2)
- Institute of Anatomy (2)
- Kidney (2)
- Principal cells (2)
- S100A4 (2)
- Thiazide (2)
- kidney (2)
- purinergic signalling (2)
- (1) ENaC activation in CCD is secondary to hyperaldosteronemia (1)
- (1) the mechanism of ENaC activation was compared in nephrotic and sodium-depleted rats (1)
- (2) recruitment of ENaC to the apical membrane determined by immunohistochemistry (1)
- (2) sodium retention and induction of Na⁺ (1)
- (3) shift in the electrophoretic profile of Γ-ENaC (1)
- 11β-hydroxysteroid dehydrogenase type 2 (1)
- Ascites (1)
- Cirrhosis (1)
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- Differentiation (1)
- Distal tubule (1)
- Edema and ascites in nephrotic syndrome mainly result from increased Na⁺ reabsorption along connecting tubules and cortical collecting ducts (CCD). In puromycin aminonucleoside (PAN)-induced nephrosis (1)
- Epithelial mesenchymal transition (1)
- Fetal growth (1)
- Fibroblast (1) Di più Meno